The Goals Were To:
• Quickly establish superior HPTA function.
• Control and suppress estrogen activity and production to prevent fat accumulation and estrogenic
negative feed-back loops.
• Suppress cortisol production and inhibit catabolic activity.
• Maintain a high CP/ATP level.
• Maintain a positive nitrogen and glycogen balance.
• Retain as much strength as possible.
Obviously estrogen control was not as difficult when Frank utilized as estrogen inhibitor during his AAS
cycle. This also was not necessary when a Cortisol/Estrogen Suppression Phase followed his Max
Androgen Phase.
Estrogen Control/HPTA Regeneration Example 1
Day 1 – HCG 500iu 4xd/Clomid 50mg 2xd/Nolvadex 30mg
Day 2 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 3 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 4 – HCG 500iu 4xd/Clomid 50mg 2xd/Nolvadex 30mg
Day 5 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 6 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 7 – HCG 500iu 4xd/Clomid 50mg 2xd/Nolvadex 30mg
Day 8 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 9 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 10 – HCG 500iu 4xd/Clomid 50mg 2xd/Nolvadex 30mg
Day 11 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 12 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 13 – HCG 500iu 4xd/Clomid 50mg 2xd/Nolvadex 30mg
Day 14 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 15 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 16 – HCG 500iu 4xd/Clomid 50mg 2xd/Nolvadex 30mg
Day 17 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 18 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 19 – HCG 500iu 4xd/Clomid 50mg 2xd/Nolvadex 30mg
Day 20 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 21 – Clomid 50mg 2xd/Arimidex 0.5mg 2xd
Day 22 – HCG 500iu 4xd/Clomid 50mg 2xd/Nolvadex 30mg
Cyclofenil 400-600 mg/d was utilized by some beasts to replace Clomid in this example.
Example 1 was a fairly common HPTA stimulation/estrogen control protocol. The reader may note the
varying periods of HCG use. This was not an accident, it merely produced better results. HCG was an LH
imitator, meaning it only replaced LH/FSH levels, not initiate their production. This meant the Leydig
Cells in the testes received an immediate signal to produce androgen endogenously.
Clomid aided in kick-starting the Hypothalamus/Pituitary production of endogenous LH but look a few
days to become effective. It did so, in part, due to reducing the effects of HPTA suppression via inhibition
of estrogenic activity upon the hypothalamus and pituitary.
Arimidex inhibits estrogen production while Nolvadex blocks estrogen receptor-sites. This of course
block or prevents negative feed-back loops (from estrogenic activity) as well as prevent female pattern fat
deposits.
Note the 500iu 4x daily of HCG schedule. I have noted for better LH and endogenous androgen profiles
with this method and quicker HPTA function rebound. Athletes often forget that HCG is actually a female
hormone that only mimics LH in males. Too much too fast and too logn can be a bitch, (all puns intended)
Cyclofenil can replace Clomid in many cases and Teslac can replace Arimidex.
Also Menotropins (75 iu twice daily with HCG injections) is another replacement for LH or can be
utilized with HCG for a double LH kick. Normal adult male testosterone levels are 300-1000 ng/dl and
are evaluated by a blood test. Remember, different countries labs use more or less conservative reference
ranges.
Note: Zinc, magnesium, and vitamin B-6 are necessary co-factors for endogenous androgen production
and significantly elevate free or unbound testosterone levels from any source. There are several nighttime
specialty products available at health food store of this nature.
If Frank did not have legal access to the items listed in example #1, he could accomplish the HPTA
rejuvenation portion by taking “Male Mix” from Hazardous Materials daily for 21 days and eat broccoli 4
times daily (about a pound total) for some estrogen control. It actually does a fair job. Adding 15,000 mg
of Tribulas Terrestris daily (45% samponin content) can also ramp up LH levels some.
The next goal, cortisol inhibition, was fairly easy. And in some beasts cases it was done without
prescription drugs (and about 50-80% as effective as low dosages of Cytadren when evaluating value
based solely on actual results). Again, this was only when a Cortisol/Estrogen Suppression Phase did not
follow a Max Androgen Phase.
Several OTC products contain phosphatidlserine. At a dosage of 800 mg daily phosphatidlserine is said to
inhibit about 30% of cortisol production/activity, which is a start.
7-isopropoxyisoflavone (ipriflavone) is a weak partitioning agent that inhibits cortisol’s effects at the
cellular level. About 1000 mg daily is necessary. 5-Methyl-7-Methoxyisoflavone is more potent than 7-
isopropoxyisoflavone. It inhibits cortisol activity, and to some extent, its accumulation to a greater degree.
Again about 1000 mg daily is necessary and works best when stacked with 7-iso. Interesting fact is that
due to its molecule structure, 5-Methyl does have some aromatase inhibitive value as well.
Vitamin-C, 1000mg 3 times daily also inhibits cortisol formation. Ephedrine at a dosage of 25mg-50mg
three times daily significantly inhibited catabolic activity when it was stacked with any other cortisol
inhibiting goodie.
Clenbuterol too, except we are not talking about prescription drugs this time.
The issue of Action/Reaction Factors applied to OTC products as well. Of course the effectiveness of any
OTC product was highly dependant upon dosage and quality.
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